Ignite Creation Date:
2024-05-05 @ 12:02 PM
Last Modification Date:
2024-10-26 @ 9:19 AM
Study NCT ID:
NCT00228345
Status:
TERMINATED
Last Update Posted:
2013-09-05
First Post:
2005-09-26
Brief Title:
The Significance of Glucose Intolerance in the Pathogenesis of Idiopathic Axonal Polyneuropathy
Sponsor:
University of Chicago
Organization:
University of Chicago
Study Overview
Official Title:
The Significance of Glucose Intolerance in the Pathogenesis of Idiopathic Axonal Polyneuropathy
Status:
TERMINATED
Status Verified Date:
2013-09
Last Known Status:
None
Delayed Posting:
No
If Stopped, Why?:
Study Completed
Has Expanded Access:
False
If Expanded Access, NCT#:
N/A
Has Expanded Access, NCT# Status:
N/A
Acronym:
None
Brief Summary:
The purpose of this study is to determine whether impaired glucose handling abnormality in the way the body processes blood sugar can cause a neuropathy damage to the nerves
Detailed Description:
Neuropathy of undetermined etiology is a common disease that usually starts at the sixth to seventh decades It can cause significant pain and disability Previous studies have demonstrated increased prevalence of abnormal glucose handling when these patients were tested with oral glucose tolerance test OGTT On the other hand many of the neuropathy patients suffer from pain and depression and obesity and abnormal OGTT in these patients may be the result of these factors We assume that if abnormal handling of blood sugar is the cause of neuropathy these patients may have evidence of damage to other organs like eyes and kidneys as a result of abnormal blood sugar In a pilot study we will determine the incidence of subtle damage to kidneys eyes and also look for other factors associated with abnormal glucose handling in patients with neuropathy and abnormal OGTT and compare it to age matched controls with normal OGTT
Study Oversight
Has Oversight DMC:
None
Is a FDA Regulated Drug?:
None
Is a FDA Regulated Device?:
None
Is an Unapproved Device?:
None
Is a PPSD?:
None
Is a US Export?:
None
Is an FDA AA801 Violation?:
None