Ignite Creation Date:
2024-07-17 @ 11:19 AM
Last Modification Date:
2024-10-26 @ 3:34 PM
Study NCT ID:
NCT06493487
Status:
NOT_YET_RECRUITING
Last Update Posted:
2024-07-10
First Post:
2024-06-29
Brief Title:
INIT Versus IASTM In Patients With Chronic PF
Sponsor:
Cairo University
Organization:
Cairo University
Study Overview
Official Title:
Integrated Neuromuscular Inhibition Technique Versus Instrument Assissted Soft Tissue Mobilization In Patients With Chronic Planter Fasciitis
Status:
NOT_YET_RECRUITING
Status Verified Date:
2024-06
Last Known Status:
None
Delayed Posting:
No
If Stopped, Why?:
Not Stopped
Has Expanded Access:
False
If Expanded Access, NCT#:
N/A
Has Expanded Access, NCT# Status:
N/A
Acronym:
None
Brief Summary:
1 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on general pain intensity in patient with chronic plantar fasciitis
2 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on pain intensity at initial morning step in patient with chronic plantar fasciitis
3 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on pain pressure threshold in patient with chronic plantar fasciitis
4 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on active dorsiflexion ROM in patient with chronic plantar fasciitis
5 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on functional disability in patient chronic plantar fasciitis
5 Investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on functional disability in patient chronic plantar fasciitis
2 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on pain intensity at initial morning step in patient with chronic plantar fasciitis
3 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on pain pressure threshold in patient with chronic plantar fasciitis
4 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on active dorsiflexion ROM in patient with chronic plantar fasciitis
5 To investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on functional disability in patient chronic plantar fasciitis
5 Investigate integrated neuromuscular inhibition versus Instrument assisted soft tissue mobilization on functional disability in patient chronic plantar fasciitis
Detailed Description:
Plantar fasciitis PFis a degenerative syndrome of the plantar fascia resulting from repeated trauma at its origin on the calcaneus Pain is generally caused by collagen degeneration at the origin of the plantar fascia at the medial tubercle of the calcaneus It affects up to 10 of the general population Functional risk factors include tightness in Gastrocnemius soleus and weakness of intrinsic foot muscles because limited dorsiflexion of tight Achilles tendon strains the plantar fascia Plantar fasciitis PF the most common cause of heel pain it accounts for approximately 11 to 15 of foot symptoms presenting to physicians The term plantar fasciitis implies an inflammatory condition However various lines of evidence indicate that this disorder is better classified as fasciosis or fasciopathy Plantar fasciitis Main roles of plantar fascia are supporting longitudinal arch of the foot and providing shock absorptionif the tension on the plantar fascia exceeds the limits of the tissue small tears can develop in the fascia Repetitive tension and subsequent tearing can cause the fascia to become inflamed and painful Plantar fasciitis is particularly common in runners but is also noted among workers who stand for long periodsAny factor which is responsible for mechanical overloading of plantar fascia can be addressed as risk factors obesity foot arch decrease dorsiflexion ROM and tightness in calf muscles One of the most common cause for limited ankle dorsiflexion range of motion ADFis gastrocnemius muscle tightness The classic presentation of plantar fasciitis is pain on the sole of the foot at the inferior region of the heel Pain is particularly bad with the first few steps taken on rising in the morning or after an extended refrain from weight-bearing activity Often the pain diminishes after a few steps and through the course of the day but returns if intense or prolonged weight bearing activity is carried out Initially the heel pain may be diffuse or migratory however with time it usually focuses around the area of the medial tuberosity of calcaneum Plantar heel pain is associated with impaired health-related quality of life including social isolation a poor perception of health status and reduced functional capabilitiesmyofascial trigger points MTrPs in the calf muscle increase the stiffness and may reduce the dorsiflexion range of ankle joint which is one of the risk factor of plantar fasciitis
Myofascial trigger points have the potential to create pain limit ROM and restrict functional activities and should therefore be addressed as part of a comprehensive physical therapy program Currently a large variety of both manual and non-manual interventions exist for the deactivation of trigger points TrPs Manual approaches may include muscle energy techniques METs strain-counterstrain SCS myofascial release proprioceptive neuromuscular facilitation and ischemic compressionIntegrated neuromuscular inhibition technique INIT is a method that includes three maneuvers in one The three techniques are ischemic compression IC or trigger point release strain counterstrain technique and muscle energy technique MET In trigger point release compression is given at the trigger point region and maintained for 15 seconds while in strain counterstrain technique the superficial fascia is stretched MET works on the principle of reciprocal inhibitionInstrument-Assisted Soft Tissue Mobilization IASTM is uses specifically designed instruments to identify and treat myofascial restrictions It is based off the principles of deep transverse friction massage It is also known as Graston Technique There are 6 stainless steel instruments which are specific for different regions and types of muscles which need to be targeted It is designed to reduce fatigue of the clinician39s hands and to detect lesions by amplifying the resonance felt through the instrument
Myofascial trigger points have the potential to create pain limit ROM and restrict functional activities and should therefore be addressed as part of a comprehensive physical therapy program Currently a large variety of both manual and non-manual interventions exist for the deactivation of trigger points TrPs Manual approaches may include muscle energy techniques METs strain-counterstrain SCS myofascial release proprioceptive neuromuscular facilitation and ischemic compressionIntegrated neuromuscular inhibition technique INIT is a method that includes three maneuvers in one The three techniques are ischemic compression IC or trigger point release strain counterstrain technique and muscle energy technique MET In trigger point release compression is given at the trigger point region and maintained for 15 seconds while in strain counterstrain technique the superficial fascia is stretched MET works on the principle of reciprocal inhibitionInstrument-Assisted Soft Tissue Mobilization IASTM is uses specifically designed instruments to identify and treat myofascial restrictions It is based off the principles of deep transverse friction massage It is also known as Graston Technique There are 6 stainless steel instruments which are specific for different regions and types of muscles which need to be targeted It is designed to reduce fatigue of the clinician39s hands and to detect lesions by amplifying the resonance felt through the instrument
Study Oversight
Has Oversight DMC:
None
Is a FDA Regulated Drug?:
False
Is a FDA Regulated Device?:
False
Is an Unapproved Device?:
None
Is a PPSD?:
None
Is a US Export?:
None
Is an FDA AA801 Violation?:
None